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"Beta Blocker" Blood Pressure Medication Modifies Expression of a Gene Involved in Heart Failure

Today lets talk about a subject near and dear to my heart. Blood pressure medication. Many years ago when I was a child I was diagnosed with hypertension (high blood pressure), I went through a battery of tests to determine a cause for my disease however none was able to be identified. Through the years since then physicians have continued to occasionally try to look in new areas in their quest to figure out why my blood pressure is as high as it is.

I have my doubts they ever will, who knows what precisely is the problem. Nevertheless, high blood pressure if left uncontrolled puts a lot of strain on my arteries and organs, so I must take daily medication to keep it in a normal range (please don't tell me that diet and exercise is really all that I need, do you think I haven't tried that? ONLY medication is able to keep things under control). One such compound (I take more than one) that is used to do this is called a beta blocker. As a result, I pay special attention to research that goes on with regards to these types of compounds and today's article is one that caught my eye!

Today let us discuss a work published in the journal Nature: Scientific Reports titled "Heart Failure and MEF2 Transcriptome Dynamics in Response to β-Blockers."

Disclaimer: THIS IS GOING TO BE A LONG ARTICLE What Is A Beta-Blocker?

A beta blocker is any such compound that is a competitive agonist against the binding of the hormones adrenaline or noradrenaline to receptors called androgenic beta receptors. Okay, lets repeat that but in a less sciency way... there are receptors that adrenaline and other similar molecules interact with, these receptors are called androgenic beta receptors (androgenic refers to the compounds that bind, the adrenaline and noradrenaline). When adrenaline binds to the beta receptor it causes a response (increased heart rate, higher blood pressure etc...). Beta blockers are compounds which bind to these receptors to...

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